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Lamivudine is indicated as part of antiretroviral combination therapy for the treatment of Human Immunodeficiency Virus (HIV) infected adults and children. Lamivudine is a prescription medicine approved by the U.S. Food and Drug Administration (FDA) for the treatment of HIV infection in adults and children 3 months of age and older.

Other names for this medication:
Lamivudine, Epivir, Heptovir, Lamivudina, Lamzid, Zeffix,

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This drug is used with other HIV medications to help control HIV infection. It helps to decrease the amount of HIV in your body so your immune system can work better. This lowers your chance of getting HIV complications (such as new infections, cancer) and improves your quality of life. Lamivudine belongs to a class of drugs known as nucleoside reverse transcriptase inhibitors-NRTI. Lamivudine is not a cure for HIV infection. To decrease your risk of spreading HIV disease to others, do all of the following: (1) continue to take all HIV medications exactly as prescribed by your doctor, (2) always use an effective barrier method (latex or polyurethane condoms/dental dams) during all sexual activity, and (3) do not share personal items (such as needles/syringes, toothbrushes, and razors) that may have contacted blood or other body fluids. Consult your doctor or pharmacist for more details. OTHER This section contains uses of this drug that are not listed in the approved professional labeling for the drug but that may be prescribed by your health care professional. Use this drug for a condition that is listed in this section only if it has been so prescribed by your health care professional. This medication may also be used in combination with other HIV medications to reduce the risk of getting HIV infection after contact with the virus. Consult your doctor for more details. A lower-strength lamivudine product is used for hepatitis B infection in people without HIV infection.

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Active TGFbeta in tissues adjacent to the injured artery did not change. The acute scrotum had resulted from spreading perianal infection due to erosion of the circlage wire. Three different glial boundaries were defined: Two of these are revealed as changes in the distribution of vimentin-immunoreactive profiles occurring in the prechiasmatic optic nerve and at the threshold of the optic tract, respectively, and one by the presence of glial fibrillary acidic protein (GFAP)-immunoreactive profiles at the chiasmatic midline. In this study, we analyzed the role of this pathway during insulin-like growth factor II (IGF-II)-induced myoblast differentiation.

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Study on drug delivery of the 3H-BCNU polylacties acid released agents on gliosarcoma UV cross-linking experiments indicated the presence, in SN17 nuclear extracts, of a prominent protein species (approximately 50 kDa) that interacts specifically with the 19-base pair element. Cerebral embolism as a complication of aneurysmectomy of the descending aorta Heart morphology differences induced by intrauterine growth restriction and preterm birth measured on the ECG at preadolescent age.

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Medline, EMBASE, and Cochrane libraries were systematically searched and 23 relevant studies identified for analysis. Factors associated with exercise lifestyle--a study of monozygotic twins. A subsystems-based approach to the identification of drug targets in bacterial pathogens. Moreover, the PH3 angiography phantom was also used to mimic human soft tissues in the brain.

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The Aldo-Keto Reductase AKR1B10 Is Up-Regulated in Keloid Epidermis, Implicating Retinoic Acid Pathway Dysregulation in the Pathogenesis of Keloid Disease. In contrast, this hydrolytic degradation cannot be found in interstitially localized macrophages.

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We demonstrate that many of the ophthalmological findings that have been considered diagnostic of abusive head trauma can also occur in association with infective processes. This paper reviews major national and international programs working towards sustainable communities, in order to arrive at strategies that establish the necessary interconnectedness and collective action within each individual community. The expression of all immunoglobulins (IgG, IgG1, IgG2 and IgE) was higher in symptomatic dogs than in all other categories.

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